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Nuclear Receptor Signaling Atlas | Receptors, Coactivators, Corepressors and Ligands
Title: Nuclear Receptor Signaling Atlas | Receptors, Coactivators, Corepressors and Ligands
Title abbreviated: NURSA
Creator: McKenna, Neil; Lanz, Rainer
Publisher: NIDDK = National Institute of Diabetes and Digestive and Kidney Diseases
Abstract: The Nuclear Receptor Signaling Atlas (NURSA) was created to develop a comprehensive understanding of the structure, function, and role in disease of nuclear receptors. While the Atlas was begun as a Functional Atlas of Orphan Nuclear Receptors to elucidate the roles played by orphan nuclear receptors in metabolism and the development of metabolic disorders (including type 2 diabetes, obesity, osteoporosis, and lipid dysregulation), as well as in processes of aging and hormone-dependent cancers, the present goal of the Atlas consortium is to expand this effort to encompass all nuclear receptors. To accomplish these objectives, the members of the consortium form a team dedicated to catalyzing progress in understanding of the complex interplay between and among nuclear receptors and coregulators, with the goal of expediting translation of basic findings into tools that can find clinical applications. The goal is to approach research questions that require complex technologies which are not easily performed in an individual investigators laboratory, and in an interdisciplinary mode. The intent is to accumulate validated data, and to make it available rapidly to interested scientists at large via electronic communication on the NURSA website. Through this website, the Atlas is designed to reach out to all interested members of the nuclear receptor research community to provide new findings, access to new reagents, large validated data sets, a library of annotated prior publications in the field, and our e-journal, Nuclear Receptor Signaling. [Information of the supplier]
Table of contents: Nuclear Receptor Signaling / NRS Articles / NURSA Publications / Datasets / Reagents / E-journal / Meetings
Subject: Metabolism in biochemical genetics (572.84)
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Audience: Experts
Language: English
Format: website; database
Resource type: Factual databases;
Discipline based websites
Access: free
Metadata update date: 2011-08-29
Metadata provider: UBFfm
URL of this vifabio-resource:
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